| Reports of presence of ENK in: PVHmpd. |
| Physiological condition | Cell pool position in region | Hemisphere | Average labeled cells | Standard deviation | Relative to basal | Annotation | Reference | Collator |
| Chemical treatment: polyethylene glycol | dorsal | bilateral | 426 | 9.99 | significantly higher | In animals injected with PEG, there was a significant increase in the number of cells containing pENK mRNA (Table 3), with the majority appearing in the dorsal part of the PVHmpd. | Watts, A.G. & Sanchez-Watts, G., 1995 | Metadata |
| in situ hybridization data |
Reports of molecules that coxepress with ENK in PVHmpd
| Coexpressed molecule | Physiological condition | Hemisphere | Cell population position | Average labeled cells | Standard deviation | Percentage cells/ENK labeled cells | Standard deviation | Percentage ENK labeled cells/cells | Standard deviation | Relative to basal | Annotation | Reference | Collator |
| CRH | Chemical treatment: polyethylene glycol | bilateral | unclear | 100 | 28.00 | 36.80 | 4.80 | 26.50 | 4.80 | not measured | After PEG injection, both the number of cells containing pENK mRNA and the number of Dig-CRH-labelled cells were significantly increased to about 37%. | Watts, A.G. & Sanchez-Watts, G., 1995 | Metadata |
| NT | Chemical treatment: polyethylene glycol | bilateral | unclear | 40 | 14.00 | 20.10 | 3.70 | 38.50 | 0.56 | not measured | In control animals, no ENK labeled cells could be detected with the Dig-pENK cRNA probe. In PEG-injected animals, substantial numbers were seen, about 20% of which also contained mRNA for NT/N (Table 3). | Watts, A.G. & Sanchez-Watts, G., 1995 | Metadata |
| in situ hybridization data |
